How to use medication for uterine fibroids? What are the causes of uterine fibroids?

What medicine should I take for uterine fibroids? We should grasp this point. Medication is a common treatment method and is of great concern to patients. What medicine should I take for uterine fibroids? The following are specific suggestions from experts.

Uterine fibroids can be treated with antagonistic hormone drugs, but there is no significant difference in the sex hormones in the blood of patients with fibroids and women without fibroids. This shows that the occurrence of fibroids is related to the hormonal environment of patients with fibroids, rather than the abnormal local endocrine environment. For example, the estrogen concentration in fibroids is higher than that in uterine muscles, and endometrial hyperplasia is higher than that near fibroids. The same is true for receptor status. E2R (estradiol receptor) and pR in fibroids are higher than those in uterine muscles.

From the perspective of histogenesis, uterine fibroid cells have long been thought to be derived from smooth muscle cells of the uterine muscle and vascular wall, such as immature myoblasts, but the latter have no clear concept histologically.

Histological studies have found that small uterine fibroids that have not grown for a long time not only have mature smooth muscle cells rich in myofilaments, but also immature smooth muscle cells found in the fetal uterus. This shows that the occurrence of human uterine fibroids may come from the differentiation process of undifferentiated mesenchymal cells into smooth muscle cells. Multiple uterine fibroids may be due to the multifocal lurking of the origin cells in the myometrium. This undifferentiated mesenchymal cell is the original cell of the fibroid, which is a cell with multiple differentiation functions in the embryonic period. It has biological media, relies on estrogen for proliferation, and relies on progesterone for differentiation and hypertrophy. After entering the sexual maturity period, the undifferentiated mesenchymal cells and immature smooth muscle cells remaining in the myometrium appear self-perpetuating under the action of the estrogen and progesterone cycles before the formation of fibroids, and repeatedly undergo the process of proliferation, differentiation and hypertrophy for a long time.