What causes adenomyosis?

The development of adenomyosis and adenomyomas is related to a variety of factors, including genetics, fluctuations in hormone levels, and repeated injuries to the uterus. Adenomyosis is a disease caused by the invasion and growth of endometrial tissue into the myometrium, while adenomyomas mean that this abnormal endometrial tissue forms one or more nodules. Conditions such as endometriosis, menstrual abnormalities, and hormonal changes may also increase the risk of these diseases.

Factors such as excessive uterine contractions, repeated childbirths, and a history of surgery may lead to damage to the myometrium and provide a pathway for endometrial tissue to enter. Fluctuations in estrogen levels in the body also play an important role in the progression of the disease, as estrogen stimulates the growth of the endometrium. Lifestyle factors, such as stress and poor diet, may also indirectly affect the onset and progression of the condition.

Factors such as excessive uterine contractions, repeated childbirths, and a history of surgery may lead to damage to the myometrium and provide a pathway for endometrial tissue to enter. Fluctuations in estrogen levels in the body also play an important role in the progression of the disease, as estrogen stimulates the growth of the endometrium. Lifestyle factors, such as stress and poor diet, may also indirectly affect the onset and progression of the condition.

If you have symptoms of uterine abnormalities such as severe dysmenorrhea, heavy menstruation or irregular bleeding, you should pay attention to it as soon as possible. Maintaining a healthy lifestyle can greatly improve uterine health, such as regular exercise to promote blood circulation and a low-fat, high-fiber diet to balance hormone levels in the body. It is also important to manage stress well, and you can consider practicing deep breathing or meditation. If the symptoms affect your daily life, it is recommended to seek medical attention in time for professional diagnosis and treatment.