What are the causes of ovarian cysts? Are these four main causes worth paying attention to? 1. Immune theory: Some people believe that when women have normal immune function, menstruation enters the local immune system of the abdominal cavity through the fallopian tube. If the local immune function is insufficient or the number of endometrial cells in the reflux abdominal cavity is too large, the immune cells are not enough to kill them, which is endometriosis. There are also reports that patients with endometriosis are twice as likely to have a history of lupus erythematosus or other autoimmune diseases. 2. Genetic theory: The incidence of the same disease in the family members of some endometriosis patients is higher than that of ordinary women, so it is speculated that there may be a genetic gene. 3. Koninckx et al. theory: Recently, Koninckx et al. have found that patients with endometriosis often have luteinized unruptuted follicle syndrome, or LUFS. Due to LUFS, the follicles are not ruptured, and the concentration of estrogen and progesterone in the ascites is low, which is conducive to the implantation of endometrial cells and prone to endometriosis. 4. Hypothalamic-pituitary-ovarian axis dysfunction: Hypothalamic-pituitary dysfunction is mainly manifested by excessive LH secretion in patients, no cyclical changes and changes in LH peak, while FSH secretion is normal or slightly low, resulting in an increase in the normal or slightly low LH/FSH ratio. LH directly acts on the ovarian theca cells P450c17a activity in the ovarian theca cells by increasing intracellular branch-chain cleavage to produce excessive androgens. |
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